People under 110 mg didn't live as long according to my research. Whatever my doctor says is suspect. Thanks.
@vadimesharak726
2 ай бұрын
Early on, ChatGPT use to show weighted answer. Then, the algorithms on sensitive topics were corrected. No way to get the initial weighted answer again. The same is about all others GPT's. But! Local LLM that will be fed with pubmed docs will answer better. Easy to prove
@sidsnyder8043
2 ай бұрын
I can manipulate my LDL levels through changes in diet, and fasting. Which tells me that lipo protein levels are controlled by your genes. It only makes sense that if you are on a high fat / low carb diet, your genes will increase production of LDL for transporting fats and cholesterol. If you are on a low fat / high carb diet, your genes will down regulate the production, since not as much LDL is needed in the body. As well, I can have my lipids checked, then go on a 5 t0 12 day fast, and on the recheck, my LDL will increase, because my body will go into fat burnning mode and needs more LDL for transport.
@btudrus
2 ай бұрын
"Which tells me that lipo protein levels are controlled by your genes." I would rather say that lipoprotein levels are controlleb by your metabolism. Agree otherwise...
@leonfuerst4790
2 ай бұрын
Russ, thanks for the video. Trust the manufacturer studies? I am glad I never started statins.
@cwebr789
2 ай бұрын
It's all about profit. Pharmaceutical companies want to sell drugs. Our well being is not what matters to them.
@avid6186
2 ай бұрын
According to the nhanes study anything under 70 is definitely making things worse. Somewhere around 130-150 is optimal in that data.
@tallgrass9366
2 ай бұрын
Thanks for bringing up chatgpt to our viewer attention. And including it in this video process. And your commentary about it.
@user-ps2nn5pj4g
2 ай бұрын
Recent discovery (on my part): high-dosage thiamine supplementation for 6 months shows improved metabolic function, including higher HDL, and lower LDL-C and triglycerides. But then, that won't sell medications, so...
@avid6186
2 ай бұрын
The very beginning of the answer might be key. "For those at high risk". What determines high risk? Thing like diabetes or smoking for instance. If you still have diabetes rather than fixing it, or still smoke rather than stopping you are still at high risk if that was the cause of your issues. But what if you fix those risk factors? Are you still at high risk?
@user-qj2wb7ne4e
2 ай бұрын
Ask your Doctor the role of LDL. Be prepared to enlighten him. Regardless of your LDL, ask your doctor what the Ratio of HDL:Triglycerides represents. Be prepared to enlighten him. What is the culprit: Lipids or inflammation caused by lifestyle? For secondary prevention I take 5mg of rosuvastatin 2x per week for inflammation not LDL reduction.
@DCGreenZone
Ай бұрын
Nattokinase will buff that plaque right out.
@avid6186
2 ай бұрын
Years ago I had a statistician tell me that for a statistis to be truly useful it needed to be independent, which most aren't. In this case you need someone needing secondary prevention who takes a statin and does nothing else. No change of diet, no change in exercise, no change in smoking and drinking habits, no change I other meds etc. I suppose you also need to also have no change in things like insulin resistance. Once you find a patient that meets those criteria you might will be able to say that statins were definitely the cause of the changes observed.
@jaroslawprokop6943
Ай бұрын
Unless you have T2D LDL is absolutely meaning less.
@dacisky
2 ай бұрын
Do one on plaque stabilizing.Anyway,excellent video as always.
Пікірлер: 20